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. 2018 Dec 29;18:197. doi: 10.1186/s12890-018-0769-3

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 8 — The hypothesized etiology of insufficient angiogenesis in pulmonary endarterectomy specimens. Insufficient angiogenesis in the organized thrombi might consist of complex conflicts between anti-angiogenic factors (decreased mRNA expression of angiogenetic factor like KDR, loscal anti-angiogenic cytokines) and pro-angiogenic factors (plasma microparticles). The finding of pro-angiogenic factors in the thrombus vessels in the present study (endothelial cells with high angiogenic potential) may represent the latter