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. 2018 Oct 23;17(1):247–255. doi: 10.3892/ol.2018.9608

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Copyright: © Su et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 2. — Large-scale training plasma samples of patients with GC (n=81) and healthy participants (n=77) of candidate RNAs. (A) Scatter plots of plasma levels of regulator of G-protein signaling 18. (B) Scatter plots of plasma levels of integral membrane protein 2B. (C) Scatter plots of plasma levels of pro-platelet basic protein. (D) Scatter plots of plasma levels of nucleosome assembly protein1-like 1. (E) Scatter plots of plasma levels of n324674. (F) Scatter plots of plasma levels of ENST00000442382. Expression levels of RNAs (2^−ΔΔCq scale y-axis) were normalized to that of the 18S rRNA gene. RGS18, regulator of G-protein signaling 18; ITM2B, integral membrane protein 2B; PPBP, pro-platelet basic protein; NAP1L1, nucleosome assembly protein1-like 1.