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. 2018 Dec 18;10(1):4. doi: 10.1038/s41419-018-1207-4

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a In 37 of 40 EEC samples (92.5%), miR-200c was expressed at higher levels than that in adjacent normal tissues. b In 25 of 37 EEC samples (67.6%), PTEN was expressed at lower levels than in adjacent normal tissues. c In 25 of 27 EEC samples (92.6%), PTEN was expressed at lower levels than in adjacent normal tissues. d miR-200c, PTEN, and PTENP1 expression in 40 cases were analyzed, and showed there was a statistically correlation between their expression. e Immunohistochemistry showed that PTEN was lost in most complex atypical hyperplasia and cancer tissues. f GEO database was used to analyze the expression of miR-200c in EEC. g The expression of PTEN in 546 cases of endometrial carcinoma were analyzed in TGCA database by http://ualcan.path.uab.edu/analysis.html. h TGCA database was used to analyze the PTENP1 expression. i–j In four EEC cell lines the expression of PTEN, PTENP1, and miR-200c was detected by qRT-PCR and western blot. There was an opposite relationship between miR-200c and PTENP1/ PTEN. **P < 0.01