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. 2018 Dec 18;10(1):4. doi: 10.1038/s41419-018-1207-4

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© The Author(s) 2018

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a The expression of estrogen receptor Alpha in four EEC cell lines. b, c The upregulation of miR-200c and downregulation of PTEN protein were disappeared after E2-induced for 48 h when stable knockdown the ESR1 in RL-952 cell lines. d, e The expression of miR-200c was increased and PTEN protein was downregulated after E2-induced for 48 h when stable overexpression the ESR1 in JEC cell lines. All results are presented as the mean of triplicate assays, with S.D. **P < 0.01