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. 2019 Jan 4;14(1):e0209196. doi: 10.1371/journal.pone.0209196

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© 2019 Yoshino et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 5 — a: MB49 cells were incubated with liposome labeled with the green fluorescence marker NBD-cholesterol for 10 min. Internalization of liposome was assessed by flow cytometry. Lip-kMA(D12) was internalized into MB49 cells in a manner similar to Lip-kMA(D22). Gray line: Liposome without NBD fluorescence. Blue line: Liposome with dendrimer D12. Red line: Liposome with dendrimer D22. b: C57BL/6 mice were administered MB49 cells and treated with each reagent. At day 28, the growth of the MB49 tumors was significantly suppressed with Lip-kMA compared to Lip-kMA(D12) (n = 5–6, p = 0.04). Values are the mean±SEM. c: Structure of C18 dialkyl dendrimers. Dendrimer D12 (C₅₁H₁₀₁N₇O₃, M.W.860.4). d: Structure of C18 dialkyl dendrimers. Dendrimer D22 (C₇₁H₁₄₁N₁₅O₇, M.W.1317).