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. 2018 Nov 27;19(2):1040–1048. doi: 10.3892/mmr.2018.9699

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 10. — Effect of a Nrf2 agonist on GSH-PX, GSH, SOD and MDA levels in PC12 diabetic model cells treated with SRT1720. ELISA was performed to determine the levels of (A) GSH-PX, (B) GSH, (C) SOD and (D) MDA in control, SRT1720-treated and SRT1720 + Nrf2 agonist-treated PC12 diabetic model cells. Curcumin was employed as the Nrf2 agonist. **P<0.01 vs. control group; ^##P<0.01 vs. SRT1 group. Nrf2, nuclear factor erythroid 2-related factor 2; GSH, glutathione; GSH-PX, glutathione peroxidase; SOD, superoxide dismutase; MDA, malondialdehyde; Con, control; SRT1, 50 mg/kg SRT1720; SRT1 + Nrf2 agonist, 50 mg/kg SRT1720 + 25 µM curcumin.