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. 2018 Nov 29;19(2):935–942. doi: 10.3892/mmr.2018.9711

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Copyright: © Wang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — Knockdown of endogenous HMGA2 suppresses cell proliferation and migration in liver cancer cells. (A) HMGA2 siRNAs (siHMGA2-1, siHMGA2-2) or negative control siRNA were transfected into Huh-7 cells. After 48 h, cells were harvested for reverse transcription-quantitative polymerase chain reaction analysis. (B) MTT assay of Huh-7 cells transfected as above. (C) Wound-healing assay of Huh-7 cells transfected as above. **P<0.01; ***P<0.001. miR, microRNA; HMGA2, high mobility group AT-hook 2; siRNA, small interfering RNA; NS, not significant.