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. 2018 Dec 12;19(2):1117–1127. doi: 10.3892/mmr.2018.9751

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Copyright: © Liang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 3. — Lv-shRNA-mediated knockdown of RPS15A regulates the growth, proliferation and apoptosis/necrosis of 786-O cells. (A) Following 3 days of incubation with Lv-shRNA or Lv-Ctrl, 786-O cell growth was recorded in cell fluorescence; magnification, ×200. (B) Celigo counting and (C) the MTT assay on 5 consecutive days. (D) Apoptotic rates of transfected 786-O cells at day 5. The results demonstrated that the percentage of apoptotic and necrotic cells was significantly higher shRPS15A compared with the shCtrl group; *P<0.05, ***P<0.001. Ctrl, control; Lv, lentivirus; OD, optical density; RPS15A, ribosomal protein S15A; shRNA, short hairpin RNA.