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. 2018 Dec 12;19(2):1117–1127. doi: 10.3892/mmr.2018.9751

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Copyright: © Liang et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 4. — Knockdown of RPS15A inhibits 786-O xenograft formation and growth in vivo. (A) Mice were divided into two groups: Group 1, The Lv-shRPS15A, and Group 2, Lv-shCtrl group. (B) Images of the tumours. (C) Mean estimated tumour volume of the Lv-shRPS15A group was significantly lower compared with the Lv-shCtrl group. (D) The average tumour weight of the xenografts from the Lv-shRPS15A group was significantly reduced compared with the Lv-shCtrl group in the seventh week. Data are presented as the mean ± standard deviation; *P<0.05. Ctrl, control; Lv, lentivirus; RPS15A, ribosomal protein S15A; sh, small hairpin RNA.