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. 2018 Nov 26;10(2):214–222. doi: 10.3892/mco.2018.1774

Table I.

Associations between STMN1, E-Cad and VIM expression and clinicopathological characteristics of GC.

STMN1 E-Cad VIM



Variables Cases (n) Positive Negative P-value Positive Negative P-value Positive Negative P-value
Sex 167 106 61 0.311 48 119 0.184 62 105 0.383
  Male 120 79 41 31 89 47 73
  Female 47 27 20 17 30 15 32
Age (years) 0.406 0.557 0.731
  <60 86 52 34 23 63 33 53
  ≥60 81 54 27 25 56 29 52
Tumor size (cm) 0.694 0.271 0.653
  <5 69 45 24 23 46 27 42
  ≥5 98 61 37 25 73 35 63
Histological differentiation 0.061 0.014a 0.408
  Well/moderate 41 21 20 18 23 13 28
  Poor/undifferentiation 126 85 41 30 96 49 77
Lauren's classification 0.037a 0.017 0.581
  Intestinal type 47 24 23 18 29 19 28
  Diffuse type 120 82 38 30 80 43 77
Serosal invasion 0.019a 0.007b 0.534
  Negative 31 14 17 15 16 10 21
  Positive 136 92 44 33 103 52 84
Lymph node metastasis 0.005b 0.0001c 0.0001c
  N0 42 19 23 21 21 16 73
  N (+) 125 87 38 27 98 46 32
TNM stage 0.008b 0.0001c 0.039a
  I+II 48 23 25 24 24 12 36
  III 119 83 36 24 95 50 69
a

P<0.05

b

P<0.01

c

P<0.0001. STMN1, Stathmin1; E-Cad, E-cadherin; VIM, vimentin; GC, gastric cancer; TNM, tumor-node-metastasis.