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. 2019 Jan 10;19:15. doi: 10.1186/s12906-018-2422-3

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© The Author(s). 2019

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 8 — Intratracheal eCS reduces the mortality of mice caused by sepsis. C57BL/6 (n = 10/group) received a single i.p. PBS () or of LPS/d-(+)-galactosamine hydrochloride, without () or with a single i.t. eCS (0.1 mg/kg body weight) 2 h later (). Morbidity and mortality were monitored for 8 days. The results are represented by Kaplan–Meier survival curves (log-rank test, * P < 0.05)

Inline graphic — Intratracheal eCS reduces the mortality of mice caused by sepsis. C57BL/6 (n = 10/group) received a single i.p. PBS () or of LPS/d-(+)-galactosamine hydrochloride, without () or with a single i.t. eCS (0.1 mg/kg body weight) 2 h later (). Morbidity and mortality were monitored for 8 days. The results are represented by Kaplan–Meier survival curves (log-rank test, * P < 0.05)