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. 2019 Jan 4;12:498. doi: 10.3389/fncel.2018.00498

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Copyright © 2019 Xu, Xing, Huang, Zhou, Liu, Cui, Cheng, Wang, Zhou, Yang, Yang, Zhang, Zang, Ma and Guan.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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HDAC1-silenced MSCs transplantation attenuated oxidative stress and neuroinflammation in the hippocampus of TBI mice. (A) Representative HEt staining on brain sections of different groups at 3 days after TBI. HEt levels in injured cortex. Scale bars = 100 μm. (B) Quantification analysis of HEt fluorescence intensity. (C) Enzyme-linked immunosorbent assay (ELISA) analysis of SOD, GSH, GSH-Px and MDA at 3 day after TBI. (D) ELISA analysis of TNF-α, IL-1β, IL-10, and IL-4 at 3 day after TBI. Data are presented as mean ± SEM. *p < 0.05 vs. Vehicle, ^#p < 0.05 vs. MSCs.