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. 2019 Jan 14;19:15. doi: 10.1186/s12935-019-0729-x

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© The Author(s) 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 1 — Bioinformatics analysis of CCNB1 in HCC tissues and normal liver tissues using TCGA cohort public database. a Comparison of CCNB1 mRNA levels in HCC tissue and non-tumor tissues in the TCGA database. The expression of CCNB1 was normalized using a logarithm. p < 0.0001, n = 424. b Comparison of CCNB1 mRNA levels in 50 pairs of HCC tissues and adjacent non-tumor tissues. c Pearson correlation between CCNB1 mRNA level and proliferation marker Ki67 mRNA level. p < 0.0001, n = 374. d Kaplan–Meier analysis of the overall survival and disease-free survival time of patients with high or low CCNB1 expression, n = 313. e The expression of CCNB1 mRNA level in HCC patients with different TNM stages (p = 0.015, n = 292)