(
A) 10X view of immunostaining in Ascl1 +CreGfp/delCreGfp samples. (
B) Corresponding GFP staining (for CREGFP fusion protein) for
Figure 5C. Even though iN cells are expressing high levels of CRE, which should loop out the Dnmt3a locus, cells are still expressing DNMT3A protein. (
C) Genotyping of Dnmt3a/b fl/fl cells. Floxed mutant bands (FL) appear at ~200 bp while deleted bands (DEL) appear at ~250 bp. (
D) Chi-squared test for significance for
Figure 5—figure supplement 1E–F. The null hypothesis is that if Dnmt3a- cells reprogram as well as the Dnmt3a + cells, there should be no significant difference in the frequency of Dnmt3a- cells and the frequency of cells that actually reprogram into neurons. There is a significant difference for both Cre (p=2.27E-6) and delCre (p=4.51E-4) conditions, indicating that Dnmt3a- cells are less efficient in reprogramming into iN cells. (
E and F) Average counts of DAPI +cells (
E) or TUJ1 +iN cells (
F) 14d post-Ascl1 induction that are either DNMT3A positive (red) or negative (gray) per 10X field of view. Using a Chi-squared test (
Figure 5D;
Figure 5—figure supplement 1D), there is a significant difference between the frequencies of total number DNMT3A- cells compared to the number of DNMT3A- iN cells in both Cre and delCre conditions.