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. Author manuscript; available in PMC: 2019 Jan 21.
Published in final edited form as: Mol Cell Neurosci. 2016 Aug 24;76:21–32. doi: 10.1016/j.mcn.2016.08.008

Fig. 1.

Fig. 1.

Up-regulation of KCa3.1 in the brains of SAMP8 mice. (A) Western blot analysis of KCa3.1 protein expression in the cortex of 3-, 7- and 12-month-old SAMR1 and SAMP8 mice. β-actin was used as the loading control. (B) The quantification of KCa3.1 protein expression in SAMR1 and SAMP8 mice (n = 3 each group). *p < 0.05, **p < 0.01, SAMP8 mice versus age-matched SAMR1 mice. (C) Quantitative real-time PCR analysis of KCa3.1 gene expression in 7-month-old SAMR1 and SAMP8 mice cortex. Values are shown for steady-state transcripts relative to β-actin in the same preparation. Results are expressed as mean ± SEM (n = 4). **p < 0.01, versus SAMR1 mice.