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. 2019 Jan 21;10:368. doi: 10.1038/s41467-018-08069-x

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Detailed characterization of rearrangements t(4;9)(q13;q31) in 15 AciCCs identify three different breakpoint patterns. Detailed presentation of rearrangements in 15 AciCCs with t(4;9)(q13;q31), with black bars indicating genomic material included in the rearrangement. Active (H3K27ac, H3K4me3) and repressive (H3K27me3) histone marks, NR4A3 binding sites and CTCF binding sites in normal parotid gland tissue are presented for comparison. Source data for translocation breakpoints are provided as a Source Data file