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. 2019 Jan 22;10:37. doi: 10.1186/s13287-019-1143-y

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — The increased osteogenesis of BMSCs caused by catalpol treatment can be partially inhibited by a Wnt/β-catenin signalling inhibitor (DKK1). a–c The expression of osteogenic-specific and Wnt/β-catenin signalling-related genes and proteins were assessed by qRT-PCR (a) and WB (b, c) at day 7 of osteogenic differentiation. d–g Osteogenic differentiation was assessed by ALP staining (d), ALP activity assays (e) and Alizarin Red staining (f). Calcium deposition was determined by optical density analysis (g). The data were confirmed by three repeated tests. The data are presented as the means ± SD. CA, catalpol; β-catenin (T), total β-catenin; β-catenin (N), nuclear β-catenin. *P < 0.05 compared with the control group, ^#P < 0.05 compared with the catalpol treatment group