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. 2019 Jan 24;9:444. doi: 10.1038/s41598-018-36914-y

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Impact of MI on total and mitochondrial triglycerides and protein levels in heart from non-obese and obese rats. Cardiac levels of (A) total triglycerides (TGs), (B) mitochondrial triglycerides. Protein expression of (C) carnitine palmitoyl transferase 1 (CPT1A), (D) Fatty acid translocase (FAT), (E) adipose triglyceride lipase (ATGL) and (F) diacylglycerol transferase 1 (DAGT1) in control rats (CT), or rats submitted to MI fed a standard (AMI) or a high fat diet (HFD-AMI). Bar graphs represent the mean ± SEM. of 8–10 animals normalized to glyceraldehyde-3-phosphate dehydrogenase (GAPDH). *P < 0.05; **P < 0.01; ***P < 0.001 vs control group. ^† < 0.05; ^††P < 0.01; ^†††P < 0.001 vs AMI group.