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. 2019 Jan 24;9:568. doi: 10.1038/s41598-018-37071-y

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Primary Mφ in MLH co-cultures regulated the magnitude of HCV- or HCV/HIV-dependent fibrogenic gene induction in LX-2 cells. (A) Gene transcript levels in LX-2 cells from MLH co-cultures uninfected or infected with HCV and HIV, normalized to average values of individual genes from uninfected MLH co-cultures. (B) Individual values of normalized gene expression levels of five fibrogenic genes whose expression were upregulated more than 1.9 fold on average by HCV infection following 7 independent MLH co-cultures. Asterisks indicate statistically significant difference measured by one-way ANOVA: **p < 0.005; *p < 0.05.