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. 2019 Jan 25;10:431. doi: 10.1038/s41467-018-08054-4

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Cross-trait genetic correlation (r_g) analysis between cancers and non-cancer traits. The traits were divided into four categories: a Common phenotypes, b Metabolic or cardiovascular related traits, c Psychiatric traits, d Autoimmune inflammatory diseases. Pair-wise genetic correlations withstood Bonferroni corrections (228 tests) are marked with double asterisk (**), with estimates of correlation shown in the cells. Pair-wise genetic correlations with significance at p < 0.01 are marked with a single asterisk (*). The color of cells represents the magnitude of correlation