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. 2018 May;15(2):129–137. doi: 10.2174/1567202615666180516120022

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© 2018 Bentham Science Publishers

This is an open access article licensed under the terms of the Creative Commons Attribution-Non-Commercial 4.0 International Public License (CC BY-NC 4.0) (https://creativecommons.org/licenses/by-nc/4.0/legalcode), which permits unrestricted, non-commercial use, distribution and reproduction in any medium, provided the work is properly cited.

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Fig. (3) — The END models of the representative patients with Diffusion Weighted Imaging (DWI), Perfusion-Weighted Image (PWI) and Fluid-Attenuated Inversion Recovery (FLAIR) imaging. (A) The DWI at 19.7 hours after stroke onset showed an acute infarct in the left Middle Cerebral Artery (MCA) territory (97.2ml, arrow), indicating positive infarct swelling model. The follow-up FLAIR imaging showed progressed infarct and brain swelling (119.8ml). (B) The DWI at 7.6 hours after stroke onset showed an acute infarct in the posterior limb of the left internal capsule (3.5ml, arrow) and a visible perfusion defect in the CBF maps (arrowhead), representing positive small subcortical infarct model. The follow-up FLAIR imaging showed infarct growth (9.3ml). (C) The DWI at 8.2 hours after stroke onset showed an infarction in the right MCA territory (22.6ml, arrow) and larger perfusion defect (T_max > 6 seconds: 37.3 ml; arrowhead). The T_max > 6s /infarct core ratio was 1.65 suggesting a mismatch model. The follow-up FLAIR imaging showed infarct growth (31.5 ml). (D) The DWI at 9.0 hours showed multiple acute infarction (0.6 ml, arrow) in the left centrum semiovale without perfusion defect, suggesting a recurrence model. The follow-up FLAIR imaging showed infarct growth (9.0 ml).