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. Author manuscript; available in PMC: 2019 Jan 29.
Published in final edited form as: J Proteomics. 2018 Feb 7;189:67–74. doi: 10.1016/j.jprot.2018.02.003

Fig. 2.

Fig. 2.

Increased core fucosylation of ELHHLQEQ762NVSNAFLDK of ceruloplasmin (A) and ML354NTSSLLEQLNEQFNWVSR of clusterin (B) significant at the stage of liver cirrhosis. Representative LC-MRM-MS traces of the transition (920.1/1179.6; [M+3H]3+) of ML354N(HexNAcFuc)TSSLLEQLNEQFNWVSR (C) and the transition (871.4/1179.6; [M+3H]3+) of ML354N(HexNAc)TSSLLEQLNEQFNWVSR (D) of clusterin (s, significant at p < 0.05; ns, non-significant; Ctrl, control; FIB, fibrosis; CIR, cirrhosis).