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. Author manuscript; available in PMC: 2019 Jan 29.
Published in final edited form as: J Parkinsons Dis Alzheimers Dis. 2018 May 21;5(1):10.13188/2376-922X.1000025. doi: 10.13188/2376-922X.1000025

Figure 2:

Figure 2:

PPAR agonist rescue of i.c. STZ induced AD-type neurodegeneration. Frontal lobe slice cultures from i.c. STZ- and vehicle-treated control rat pups were treated for 72 hours with PPAR-δ + PPAR-γ agonists or vehicle (See Legend to Figure 1). Duplex ELISAs measured (A) choline acetyltransferase (ChAT), (B) acetylcholinesterase (AChE), (C) Tau, (D) pTau, (E) pTau/Tau calculated mean ratios, and (F) amyloid-beta peptide (1-42) of amyloid beta precursor protein (AβPP-Aβ) with results normalized to RPLPO. Data were analyzed by two-way ANOVA (Table 1). Significant post hoc test results are displayed.