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. 2018 Oct-Dec;10(4):4–18.

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Copyright ® 2018 Park-media Ltd.

This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 5 — (A) Microarray for the analysis of the drug resistance of RTI causative agents. The biochip contained immobilized probes corresponding to the species-specific polymorphism of the 16S rRNA gene, which were used for the identification of microorganisms, and also probes specific to the rrs, rrl, gyrA, parC, mefA, mtrR, nimB-G, penA, ponA, porB, rpsJ, ntr4tv, ntr6tv, blaSHV, blaTEM, and tetM genes sequences, which act as determinants of resistance of RTI causative agents to different AMD. The elements containing wild-type oligonucleotides are circled in black. (B) The hybridization pattern obtained by analyzing N. gonorrhoeae DNA contained the following mutations: S91F+D95G in the gyrA gene (group 2), -35delA in the promoter of the mtrR gene (group 3), insD345 in the penA gene (group 4), and S87R in the parC gene (group 10)