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. 2018 Dec 11;39:83–94. doi: 10.1016/j.ebiom.2018.12.003

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© 2018 The Authors

This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/4.0/).

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Fig. 1 — VPA treatment improved cardiac function and attenuated myocardial damage in rats at 24 h after MI. (a) Timeline of MI model generation and VPA treatment. (b) and (c) Echocardiographic measurements of ejection fraction (EF) and fractional shortening (FS) of Sham, MI, MI + VPA and MI + VPA 60 min rats at 24 h after MI. **, P < .01 vs Sham group; ##, P < .01 vs MI group; n = 9 except for MI + VPA 60 min group, which is 4. (d) Representative images of heart sections by TTC staining. Scale bar: 2.5 mm (e) Quantitative analysis of infarct size expressed as percentage of left ventricle. **, P < .01, n = 6. (f) Serum CK and (g) serum LDH levels. **, P < .01 vs Sham group; #, P < .05 vs MI group; ##, P < .01 vs MI group; n = 9. All samples were analyzed by one way ANOVA followed by post-hoc Turkey HSD analysis. Data were expressed as mean ± SEM.