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. 2018 Dec 11;39:83–94. doi: 10.1016/j.ebiom.2018.12.003

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© 2018 The Authors

This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/4.0/).

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Fig. 2 — VPA treatment decreased oxidative stress and cell apoptosis after acute MI. Myocardial MDA (a) and SOD (b) activities. **, P < .01 vs Sham group; #, P < .05 vs MI group; ##, P < .01 vs MI group; n = 6. (c) Representative images of in situ detection of apoptotic cells by TUNEL staining. Myocardial tissues were stained and analyzed with TUNEL (green) and counterstained with DAPI (blue). Scale bar: 200 μm (d) Quantitative analysis of TUNEL positive cells expressed as percentage of DAPI stained cells. **, P < .01, n = 3, N.D., No Detectable. (e) Representative images of Western blot of Bax and Bcl2 expression from Sham, MI and MI + VPA treated rats. (f) Relative intensity of Bax/Bcl2 over α-Tubulin. *, P < .05 vs Sham group; #, P < .05 vs MI group, n = 3. All samples were analyzed by one way ANOVA followed by post-hoc Turkey HSD analysis. Data were expressed as mean ± SEM.