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. 2018 Nov 28;39:377–387. doi: 10.1016/j.ebiom.2018.11.040

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© 2018 The Authors

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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Fig. 6 — GSK3β regulates GluA1 phosphorylation and HCN4 expression in the brain in established epilepsy. Hippocampi from wildtype (n = 7) and GSK3β[S9A] (n = 8) mice 21 days after the KA injection were isolated and lysed. (a) Western blot was used to determine the levels of GluA1, P-GluA1 (S845), P-GluA1 (S831), GAD67 and HCN4. Tubulin is shown as a loading control. (b) Quantitative Western blot analysis results. Error bars indicate SEM. ***p < .001 (two-way ANOVA significant interaction effect, p = .0001, ***; two-way ANOVA with Bonferroni correction).