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. 2019 Feb 1;17:20. doi: 10.1186/s12951-019-0458-2

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© The Author(s) 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 6 — a Cell viability of HUVECs; ANP promotes coagulation abnormalities based on a 3D human microvascular model: b reactive oxygen species (ROS), c malondialdehyde (MDA), d heme oxygenase-1 (HO-1), e NF-κB, f interleukin 1 (IL-1), g tissue factor, h thrombomodulin, i tissue plasminogen activator [Vc + ANP: Vitamin C-0.1 mg/ml, ANP-40 μg/ml, SP: special control-ANP (40 μg/ml) but without cells] (*P < 0.05, **P < 0.01, compared with the control, n = 5)