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. 2019 Jan 31;10:48. doi: 10.1186/s13287-019-1148-6

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© The Author(s). 2019

Open Access This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — ERK/MAPK signaling pathway was activated by TGC-CM during odontogenic differentiation of BMMSCs. a TGC-CM induced significantly high phosphorylated level of ERK in BMMSCs after incubated in TGC-CM for 0 min, 15 min, 30 min, and 60 min. But the phosphorylated level of p38 and JNK did not change. b The phosphorylated level of ERK was also increased in BMMSCs during odontogenic differentiation for 7 and 14 days