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. 2019 Feb;189(2):258–271. doi: 10.1016/j.ajpath.2018.10.007

Figure 2.

Figure 2

Depletion of hepatocyte integrin αvβ8 did not alter inflammatory phenotype after partial hepatectomy. A–C: Quantification and representative images from F4/80 (Kupffer cell; A), GR1 (neutrophil; B), and platelet-derived growth factor receptor (PDGFR)β (hepatic stellate cell; C) immunostaining of liver tissue from control and Itgb8flox/flox;Alb-Cre (β8-AlbCre) mice at 48 hours after partial hepatectomy. D: Quantitation of 5-bromo-2′-deoxyuridine (BrdU)+ hepatocyte nuclei and representative images of BrdU immunostaining of liver tissue from control and Itgb8flox/flox;Pdgfrb-Cre (β8-PdgfrbCre) mice at 48 hours after partial hepatectomy. Data are expressed as means ± SEM. n = 5 control and β8-AlbCre mice (A–C); n = 4 and 8 control and β8-PdgfrbCre mice (D). Scale bar = 100 μm.