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. 2019 Jan 14;30(2):187–200. doi: 10.1681/ASN.2018080853

Table 2.

Renal manifestations from pharmacologic VEGF inhibition in murine models

Animal Model (Model/Transgene, Strain, Age) Drug Mechanism Target Dose, Route and Frequency Effects Reference
Wild-type mice, CD1, age not specified Anti-VEGF antibody and mouse sFlt-1/Fc fusion protein (soluble VEGFR1) Both treatments act to reduce circulating VEGF VEGFA 3.25 and 32.5 pM, IV, single dose Both treatments induced proteinuria by 3 h after administration, which resolved after 24 h. Both treatments resulted in GEnC hypertrophy and detachment from the basement membrane, starting at 3 h post-treatment and persisting to 24 h 17
Wild-type mice, strain not specified, neonatal Antibody to recombinant human VEGF165 Reduction of circulating VEGFA VEGFA 100 µl/dose, IP, single dose on days 0, 2, 4, and 5 after birth Decreased glomerular number and formation of abnormal glomeruli with poor cellularity and increased ECM. No significant changes in any nonglomerular vessels 21
Wistar Kyoto rats treated with antiglomerular basement membrane Ab, 12 wk Mouse sFlt-1 plasmid Soluble decoy receptor for VEGFR1. Reduction of circulating VEGFA Soluble VEGFR1 500 μg, IM, 3 d before and 2 wk after injection of antiglomerular basement membrane Ab Accelerated renal failure, proteinuria, interstitial fibrosis, endothelial cell loss. and downregulation of Nephrin in a model of rat crescentic GN 35
Wild-type mice, C57BL/6, 9–13 wk old Axitinib (AG-013736) Small molecule multitargeted TKI against VEGFR1–3, c-KIT, and PDGFR VEGFR2, VEGFR1, VEGFR3, c-KIT, PDGFR 25 mg/kg, IP, twice daily for 7, 14 or 21 d Reduction in peritubular capillary density by 30% and glomerular capillary by 10% after 21 d of treatment. Dose dependent increase in proteinuria. Reduced glomerular capillary fenestrations. No increase in serum creatinine 51
For dose-response studies: 1, 10, or 100 mg/kg, oral gavage, twice daily for 7 d
Wild-type mice, C57BL/6, 9–13 wk old Ad-sVEGFR1 Adenoviral vector that expresses the extracellular domain of murine VEGFR1. Acts as soluble decoy receptor for VEGF Soluble VEGFR1 1×109 plaque-forming units, tail vein, once No significant reduction in peritubular capillary or glomerular capillary density. Reduced glomerular capillary fenestrations and increase proteinuria after 14 d 51
BALB/c(Bicc1/Bicc1) BPK model (murine phenocopy of ARPKD) and BALB/c wild-type controls, age not specified Tesevatinib TKI including EGFR, HER2/ErbB2, c-Src, and VEGFR2 VEGFR2, HER2, EGFR2, ERBB2 7.5 and 15 mg/kg, IP, daily postnatal day 4–21 Dose-dependent reduction in whole kidney size, total kidney weight; altered renal and liver morphology 48
PCK rat model (orthologous model of human ARPKD) and Sprague–Dawley wild type as control, age not specified Tesevatinib TKI including EGFR, HER2/ErbB2, c-Src, and VEGFR2 VEGFR2, HER2, EGFR2, ERBB2 7.5 and 15 mg/kg, oral gavage, daily for 60 d (from postnatal day 30–90) Dose-dependent reduction in whole kidney size, total kidney weight; altered renal and liver morphology 48
UUO model and folic acid nephropathy models in male wild-type C57BL/6 mice, 6–8 wk Nintedanib (BIBF11220) A multitargeted TKI that blocks PDGFR, VEGFR, FGFR, and Src family kinases PDGFR, VEGFR, FGFR, SRC 50 mg/kg, oral gavage, administered starting on day of UUO and then daily for 7 d Attenuated renal fibrosis, inhibited activation of renal interstitial fibroblasts, and suppressed expression of proinflammatory cytokines after UUO 49
db/db and db/m male C57BL/6 mice, 6 wk dRK6 (a D-amino acid derivative of RK6) An arginine-rich anti-VEGF hexapeptide that binds with VEGF-A, and blocks the interaction between VEGFA (mainly VEGF165 and VEGF121) and the VEGFRs VEGFA 50 μg, SC, three times per week starting at 8 wk of age and lasting until 12 wk (short-term) and 20 wk (long-term) Both short-term and long-term treatment had decreased creatinine clearance compared with control db/db mice. Long-term treatment also exacerbated albuminuria, mesangial matrix expansion, and glomerulomegaly as compared with vehicle-treated db/db and short-term dRK6-treated db/db mice 36
Perinatal wild-type mice (exact age not specified) DC101 mAb against the extracellular portion of the VEGFR2 VEGFR2 0.08 mg/dose, IP, on postnatal days 2 and 4 Large renal cysts, impaired glomerulogenesis (hypocellular), and increased albuminuria by 3 wk of age 50
Male Wistar Kyoto rats 280–300 g, age not specified Sunitinib A multitargeted TKI including PDGFRα and PDGFRβ, VEGFR1, VEGFR2, and VEGFR3, and FMS-like tyrosine kinase-3 VEGFR1, VEGFR2, VEGFR3, PDGFRα, PDGFRβ, FMS-like tyrosine kinase-3 Low dose (7 mg/kg), intermediate (14 mg/kg) or high (26.7 mg/kg), oral gavage, once daily for 8 d All doses associated with hypertension and proteinuria. Intermediate and high doses were associated with endothelial swelling. High-dose only was associated with fibrin deposits in glomerular capillaries and small arteries 52

IV, intravenous; GEnC, glomerular endothelial cells; IP, intraperitoneal; ECM, extracellular matrix; Ab, antibody; IM, intramuscular; c-KIT, tyrosine-protein kinase Kit; PDGFR, PDGFR receptor; ARPKD, autosomal-recessive polycystic kidney disease; PCK, polycystic kidney; UUO, unilateral ureteric obstruction; EGFR, EGF receptor; FGFR, fibroblast growth factor receptor; SC, subcutaneous.