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. 2018 Nov 27;70(3):949–961. doi: 10.1093/jxb/ery423

Fig. 5.

Fig. 5.

The pep2 mutant plants flower earlier than the wild type and show reduced reverted phenotypes. (A) Wild-type (WT) plants exposed to several durations of vernalization (8, 12, 18, and 21 weeks) followed by 3 weeks in LDs. (B) pep2-1 mutant plants exposed to several durations of vernalization (8, 12, 18, and 21 weeks) followed by 3 weeks in LDs. Scale bar=10 cm. (C) Time to flower emergence of WT and pep2-1 plants exposed to different durations of vernalization measured as the number of days to the first open flower. (D) Percentage of flowering inflorescence branches (FB) in the WT and the pep2-1 mutant exposed to 8, 12, 18, and 21 weeks of vernalization at the time the last flower in the inflorescence opened. (E) WT reverted inflorescence in plants vernalized for 8 weeks. (F) pep2-1 mutant inflorescence in plants vernalized for 8 weeks. Scale bar=2 cm. (G) Number of bracts within the inflorescence of the WT and the pep2-1 mutant exposed to 8, 12, 18, and 21 weeks of vernalization at the time the last flower in the inflorescence opened. This experiment was performed together with the pep1-1 mutant in an experiment previously published (Fig. 6 in Lazaro et al., 2018). Data for the WT control is similar between the two papers. Asterisks stand for significant differences between the wild type and the pep2-1 mutant at each time point determined by multiple pairwise Bonferroni tests (α-value of 0.05). Error bars indicate the standard deviation.