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. 2018 Dec 19;17(3):1706–1716. doi: 10.3892/etm.2018.7116

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Copyright: © Wu et al.

This is an open access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivs License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non-commercial and no modifications or adaptations are made.

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Figure 5. — Effect of PTEN on rat cardiomyocyte injury induced by septic shock serum. (A) Bioinformatics prediction of binding sites between miR-494-3p and PTEN mRNA. Western blots of PTEN in rat cardiomyocytes transfected with (B) miR-NC, miR-494-3p mimics, (C) siR-NC or siR-PTEN. Effect of PTEN overexpression on (D) proliferation, (E) cell cycle distribution, (F) apoptosis and (G) LDH expression. *P<0.05 vs. siR-NC group. Control, cardiomyocytes cultured under normal conditions; miR, microRNA; NC, negative control; siR, short interfering RNA; PTEN, phosphatase and tensin homolog; LDH, lactate dehydrogenase.