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Allicin induced SiHa cell apoptosis mainly by suppressing the expression of NRF2. (A) Western blot assay showed that allicin-inactivated PI3K/AKT signaling could be partially reversed by overexpressing of NRF2. (B) Allicin-induced cell apoptosis could largely be abolished by upregulation of NRF2. *P<0.05, **P<0.01 and ***P<0.001, vs. control. p, plasmid; NRF2, nuclear factor erythroid 2-related factor 2; AKT, protein kinase B; p-AKT, phosphorylated AKT; Bcl-2, B-cell lymphoma 2.
