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. 2019 Feb 6;38:57. doi: 10.1186/s13046-019-1070-x

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — TRIM14 function was demonstrated in vivo and IHC. a ShCtrl-transplanted and shTRIM14-transplanted LN229 cells were counted and transplanted in orthotopic nude mice model respectively, n = 6. b Survival curve of shCtrl-transplanted or shTRIM14-transplanted intracranial xenografts. ***, P < 0.001, n = 6. c Representative pseudocolour bioluminescence images of orthotopic tumours bearing control or TRIM14-depleted LN229 cells on the days as indicated. Quantification of tumor volume is shown. ***p < 0.001; n = 3 experiment. d The HE assay was performed to show tumor cytostructure. e Western blot analysis of TRIM14 and ZEB2 extracted from four pairs mice from shCtrl-transplanted group and shCtrl-transplanted group. Equal loading is confirmed by β-actin levels. f IHC staining of TRIM14 and ZEB2 in four representative GBM specimens