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. 2019 Feb 1;10:48. doi: 10.3389/fphar.2019.00048

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Copyright © 2019 Xiang, Yang, Liu, He, Zhang, Li, Zhang and Liu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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DNA microarray analysis of non-cholestatic, 17α-ethinylestradiol (EE) and EE+CBS rat livers. (A) Venn diagram analysis of differentially expressed genes of non-cholestatic, EE and EE+CBS groups. (B) Heat map of differentially expressed genes in non-cholestatic, EE and EE+CBS groups (C) gene ontology (GO) analysis of differentially expressed genes (D) Kyoto encyclopedia of genes and genomes (KEGG) pathway analysis of differentially expressed genes. Arrows point to the pathways related to bile acid (BA) homeostasis; (n = 3). Abbreviations: BP, biological process; CC, cellular component; MF, molecular function. Interpretation: GO:0042493, response to drug; GO:1903298, negative regulation of hypoxia-induced intrinsic apoptotic signaling pathway; GO:0000122, negative regulation of transcription from RNA polymerase II promoter; GO:0050821, protein stabilization; GO:0060662, salivary gland cavitation; GO:0070062, extracellular exosome; GO:0031012, extracellular matrix; GO:0005790, smooth endoplasmic reticulum; GO:0005925, focal adhesion; GO:0005794, Golgi apparatus; GO:0005515, protein binding; GO:0019900, kinase binding; GO:0042803, protein homodimerization activity; GO:0005506, iron ion binding; and GO:0004305, ethanolamine kinase activity.