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. 2019 Feb 11;14(2):e0212096. doi: 10.1371/journal.pone.0212096

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© 2019 Garcia-Bustos et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 3 — A. Detail of a vacuolated subendocardial Purkinje cell. Subacute reperfused infarction group. 400x magnification. Hematoxylin-eosin. Microvacuoles predominate in this micrograph. B. Cluster of perivascular Purkinje cells with large vacuoles. Subacute reperfused infarction group. 200x magnification. Hematoxylin-eosin. An active fibroblastic response and hemorrhage can be seen. C. Intramyocardial Purkinje and transitional cells in the acute phase. Acute reperfused infarction group. 200x magnification. Hematoxylin-eosin. Note microvascular obstruction of the capillaries. D. Necrotic subendocardial Purkinje cells and cardiomyocytes in the acute non-reperfused infarction group. 200x magnification. Hematoxylin-eosin. C, capillary; CM, cardiomyocyte; E, endocardium; e, eosinophil; m, mononuclear cell; TC, transitional cell; PC, Purkinje cell; V, vein; v, vacuoles.