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. 2019 Feb 11;14(2):e0212096. doi: 10.1371/journal.pone.0212096

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© 2019 Garcia-Bustos et al

This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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Fig 4 — A. Intramyocardial perivascular Purkinje cells. Acute reperfused infarction group. 200x magnification. Hematoxylin-eosin. Note acute hypereosinophilia of inferior cardiomyocytes. B. Small epicardial cluster of pericoronary Purkinje cells. Subacute reperfused infarction group. 200x magnification. Hematoxylin-eosin. C. Perivascular penetrating Purkinje cells. Chronic infarction group. 200x magnification. Hematoxylin-eosin. D. Cluster of perivascular vacuolated Purkinje cells. Subacute reperfused infarction. 100x magnification. Hematoxylin-eosin. Tissue necrosis and inflammatory infiltrate are evident. A, artery; CM, cardiomyocyte; PC, Purkinje cell; V, vein.