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. 2019 Feb 11;9:1732. doi: 10.1038/s41598-018-38389-3

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Glomerular podocyte injury in RK rats is attenuated by SF. (a) Western blot analysis of nephrin in the plasma membrane-enriched fraction of the kidney in RK and RK + SF rats. Bar graphs show the results of quantitation (n = 4 each group). (b) Representative micrographs of immunostaining for desmin, a marker for podocyte injury. Bar graphs show the quantitative evaluation of desmin staining in the glomeruli. Bar in the upper panel represents 100 μm. Bar in the lower panel represents 50 μm. (c) Transmission electron micrographs of podocyte foot process in the glomeruli of indicated animals. Bar represents 5 μm. Data are expressed as mean ± SEM; *P < 0.05. **P < 0.01.