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. 2019 Feb 11;19:136. doi: 10.1186/s12885-019-5337-6

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 5 — Endogenous expression of direct targets of miR-200c in mouse lung non-metastatic (KW-634, blue bars) and metastatic (821-T4, violet bars; 821-LN, green bars) cell lines, showing the various roles and effectiveness of our Nano encapsulation of miR-200c after each treatment with 50 nM miR-200c and nano-miR-200c. a- Nkx2.1 expression levels were maintained following treatment with Nano miR-200c in non-metastatic KW-634 and were elevated in metastatic 821-T4, while they were almost undetectable in metastatic 821-LN. b- Six4 showed significant expression in the cell lines treated with miR-200c and Nano miR-200c, and c- Myb expression was considerably affected by Nano miR-200c treatment in all lines