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. 2019 Feb 13;17:12. doi: 10.1186/s12964-019-0322-x

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — PAX8 suppressed thyroid carcinogenesis after LDR exposure. a Analysis of PAX8 target signature genes in BRAF^V600E transformed N-thy ori-3-1 cells after fractionated (0.01Gyx10) dose of radiation. b qPCR analysis of NIS and TG expression in LDR-treated 850-5C thyroid cancer cells after PAX8 silencing. c, d Sphere and colony formation was determined in 850-5C cancer cells in same conditions as shown in B. β-actin was used as a control for normalization. *p < 0.05, **p < 0.001, and ***p < 0.0001