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. 2019 Feb 12;13:40. doi: 10.3389/fncel.2019.00040

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Copyright © 2019 Roselló-Busquets, de la Oliva, Martínez-Mármol, Hernaiz-Llorens, Pascual, Muhaisen, Navarro, del Valle and Soriano.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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MβCD increases GAP43 expression in injured DRG neurons. (A,B) Detail of DRG (A, dashed line) and motoneurons (B, dashed line) analyzed for GAP43 expression. (C,D) Quantification of GAP43 positivity in DRG neurons (C) and motoneurons (D) from animals treated with vehicle or MβCD vs. Saline. (E) Detail of a regenerated nerve stained for cell nuclei (blue), NF-H (green), and GAP43 (red) of saline- (top) and MβCD-treated (bottom) animals. Scale bar: 15 μm in (A,B); 50 μm in (E). Student’s t-test, ^∗∗p < 0.01.