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. 2019 Feb 18;10(3):166. doi: 10.1038/s41419-019-1415-6

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a qPCR analysis of the mRNA level of PBK in high-grade serous ovarian cancer (HGSOC) tissues and normal fallopian tube (FT) tissues (FT, n = 12; HGSOC, n = 24). b Western blot analysis of the protein level of PBK in HGSOC tissues and FT tissues (FT, FT1-FT5; HGSOC, T1-T11; uncut bands were shown in Supplementary Figure S1A). c Western blot analysis of the protein level of PBK in ovarian cancer cell lines (A2780, HEY, HO8910, OVCAR3, SKOV3, HO8910PM) and normal fallopian tube epithelial cells (FTE187). d Representative images of IHC staining of PBK in HGSOC tissues and FT tissues (upper, ×40 ; lower, ×400). e The high and low expression rate of PBK in 54 FTE tissues and 234 HGSOC tissues. f Overall survival (OS) curves of HGSOC patients with high or low PBK expression level in our cohort. g Correlation between PBK expression and cisplatin resistance in HGSOC patients (data are mean ± SEM, **p < 0.01)