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. 2019 Jan 30;70(4):1239–1253. doi: 10.1093/jxb/ery456

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© The Author(s) 2019. Published by Oxford University Press on behalf of the Society for Experimental Biology.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 1. — Functional characterization of pea auxin receptors with 2,4-D in Arabidopsis. Seedlings at 4 d old were transferred to media containing 2,4-D for 3 d. The seedlings assessed were Col-0, Attir1-10, Attir1-10 afb2-3, or those expressing (A, B) pAtTIR1::cPsTIR1a (PsTIR1a-A8 and C1), pAtTIR1::cPsTIR1b (PsTIR1b-C3-2 and C4-1) or pAtTIR1::cAtTIR1 (AtTIR1-B1-2 and C2-5) in Attir1-10, or (C–E) pAtTIR1::cPsTIR1a (PsTIR1a-B1-7 and C2-12), pAtTIR1::cPsTIR1b (PsTIR1b-B1-8 and C3-6), pAtTIR1::cPsAFB2 (PsAFB2-A2-6 and B1-5) or pAtTIR1::cAtTIR1 (AtTIR1-A2-5 and B1-8) in Attir1-10 afb2-3. Root elongation is expressed as a percentage of the same genotype in medium without auxin. Data are means (±SD), n=8. Different letters indicate significant differences within the same auxin concentration as determined by one-way ANOVA and Holm–Sidak post hoc tests (P<0.05).