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. 2019 Feb 21;38:93. doi: 10.1186/s13046-019-1109-z

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© The Author(s). 2019

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — Zoledronic acid attenuates the phosphorylation of Erk1/2 and Akt in cervical cancer cells derived CSCs. Western blot analysis of MAPK- and PI3K/Akt pathways related proteins (total Erk1/2, pho-Erk1/2, pho-JNK, pho-p38, PI3K, total Akt, pho-Akt, total p38, and JNK) in cervical cancer cells derived CSCs and parental cervical cancer cells (a-c). Western blot analysis of total Erk1/2, pho-Erk1/2, pho-JNK, pho-p38, PI3K, total Akt, pho-Akt, total p38, and JNK in cervical cancer cells derived CSCs treated or not with 10, 20, and 30 μM zoledronic acid (d-f). Control vs. 10, 20, and 30 μM of zoledronic acid: * P < 0.05, ** P < 0.01. Results are shown as mean values ± SD of independent experiments performed in triplicate