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Contribution of renin–angiotensin system-mediated actions in the epileptogenesis process. Exacerbation of epilepsy pathophysiology through predominance of the Ang II/ATR1 axis, and available targeted therapeutic strategy through RAS enzyme inhibitors; ATR1 antagonists; and ATR2, MasR, MrgD, and ATR4 agonists that mediate beneficial actions in epilepsy pathophysiology and related consequences.
