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. 2018 Feb 27;18:3. doi: 10.1186/s12899-018-0037-z

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© The Author(s). 2018

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 3 — Hepatocyte-specific Alk3 deficiency resulted in elevated liver ferroportin expression 14 days after BA injection. a Relative hepatic ferroportin mRNA levels (*P = 0.01: Alk3^fl/fl; Alb-Cre injected with saline [n = 4] vs Alk3^fl/fl; Alb-Cre injected with BA [n = 6]; *P = 0.01: Alk3^fl/fl injected with BA [n = 5] vs Alk3^fl/fl; Alb-Cre injected with BA [n = 6]). Representative Western blots (b) and quantitative analyses (c) of hepatic ferroportin protein levels with α-tubulin as loading control