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. 2019 Feb 19;10:91. doi: 10.3389/fendo.2019.00091

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Copyright © 2019 Guo, Liu, Zhao, Mi, Zhang and Li.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Schematic illustration of FSH and KIT signaling in promoting primordial follicle assembly in the chicken. FSH promoted primordial follicle formation, and KIT signaling augmented the sensibility of FSHR to FSH via intracellular signal transduction. In the process of primordial follicle formation in the chickens, FSH cooperated with SCF reciprocally to promote cell proliferation, the expression of N-cadherin, and transcription factors, but also acted to inhibit E-cadherin/β-catenin and cell apoptosis.