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. 2019 Feb 26;9:2821. doi: 10.1038/s41598-019-39541-3

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© The Author(s) 2019

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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(A) Tlr2−/− mice survival after ischemic lesion is better than of their wild type (WT) counterparts analyzed by Log-rank (Mantel-Cox) test P = 0.037*; (B) Both, Tlr2−/− and WT lose body weight after ischemic lesion, and subsequently regain it after 28 days; (C) Mice that reached humane endpoint (grey and pale red) (non-survivors, NS) had worse neurological scores than their survivor counterparts. There was no significant difference between wild type (WT), and Tlr2−/− mice. (D) Bilateral tactile stimulation test revealed decreased latency in sticky tape removal from the contralateral forepaw in acute time points in Tlr2−/− mice compared to their wild type (WT) counterparts, but increased time of reaction in the later time points of 14 and 28 days. ***,** and *marking P < 0.001, P < 0.01, P < 0.05 for differences between WT controls and Tlr2−/− controls, respectively. #Are used to mark differences within the same genotype compared to their sham operated controls (WT controls vs. WT tMCAO, Tlr2−/− controls vs. Tlr2−/− tMCAO).