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. 2019 Feb 27;38:104. doi: 10.1186/s13046-019-1098-y

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© The Author(s). 2019, corrected publication 2021

Open AccessThis article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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Fig. 7 — TRIM29 mediates EMT and invasion via activating the Wnt/β-catenin signaling pathway. a Modified RKO cells were treated with XAV939 (15 μM) for 24 h, and the indicated protein levels were then assayed by Western blotting. laminB1, β-actin, and α-Tubulin were used as internal controls. b The β-catenin protein expression and localization in RKO-Vector and RKO-TRIM29 cells with XAV939 (15 μM) treatment was assessed by immunofluorescence assay. c The Transwell invasion assay showed different cell invasive abilities in RKO-Vector and RKO-TRIM29 cells with XAV939 (15 μM) for 24 h. The data are presented as the mean ± SEM. from three independent experiments (**P < 0.01, ***P < 0.001)